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Writer's pictureJoshua Francois

Progressive Collapsing Foot Deformity (PCFD) | New Terminology for Adult Acquired Flatfoot | Find Your Stride | Edinburgh Podiatrist

Introduction

Progressive collapsing foot deformity (PCFD), previously referred to as adult acquired flatfoot (AAF), is a multifaceted condition affecting the foot and ankle. This disorder leads to the flattening of the foot's arch, accompanied by various other deformities that may not be immediately apparent. A closely related condition is posterior tibial tendon dysfunction (PTTD), which is typically viewed as a significant contributing factor to the progression of PCFD. Understanding the etiology, anatomy, symptoms, and treatment of PCFD is essential for effective management and relief.


Left fallen medial longitudinal arch
A loss of integrity within the midfoot and fallen medial longitudinal arch (left) is typical of this condition

Understanding the Condition

PCFD manifests as a spectrum of disorders, commencing often with mild symptoms that can escalate into severe, debilitating flatfoot. While many individuals in the initial stages can find relief through conservative measures such as orthotics and bracing, surgical intervention may become necessary if these non-invasive treatments fail to alleviate pain and improve functional limitations.


Pathophysiology

The prevailing view is that PCFD generally stems from dysfunction of the posterior tibial tendon (PTT), a crucial structure responsible for maintaining the arch of the foot. When the tendon suffers an injury or tears, its capacity to provide stability diminishes, leading to a collapse of the arch. Over time, this dysfunction initiates a cascade of complications as other supporting structures, including ligaments, become strained and weakened.


Anatomy of the Foot

The anatomy of the foot involves various structures that contribute to arch integrity. These include:


The Posterior Tibial Tendon (PTT)

The PTT links the tibialis posterior muscle to the bones supporting the arch on the inside of the foot. This tendon winds its way behind the medial malleolus (the prominent bone on the inside of the ankle), playing a vital role in arch maintenance and foot stability during movement.


The Spring Ligament

Connecting the heel bone (calcaneus) to the navicular bone, the spring ligament is essential for arch support. As PCFD progresses, it may become stretched or torn, further contributing to the collapse of the foot's structure.


Other supporting ligaments, including the deltoid ligament, also play a role in maintaining the foot’s anatomy. Over time, as flatfoot continues to develop, even these robust ligaments can become overstretched, compounding the deformity.


Etiological Factors

There are multiple potential causes of PCFD, and these may vary among patients. Some common contributors include:


  • Mid-foot arthritis or lax ligaments that exert abnormal stress on the PTT, potentially leading to tears.

  • Acute injuries from falls that can damage the PTT or initiate inflammation.

  • Chronic overuse, especially in individuals participating in high-impact sports, may result in gradual tendon damage. Non-athletes can also encounter similar issues, particularly when wearing unsupportive footwear.

  • Genetic predisposition may play a significant role in the development of this condition.


Symptoms

Individuals with PCFD typically experience various symptoms, including:


  • Pain in the arch or along the inside of the foot and ankle, often correlating with inflammation of the PTT.

  • Weakness during activities that place strain on the foot, such as standing, walking, or participating in high-intensity sports.

  • Discomfort on the outer side of the ankle, possibly resulting from alignment changes in the heel bone due to the arch's collapse.


Nonsurgical Treatment Options

Many patients find relief through conservative treatments, which may include:


Rest and Activity Modification

Reducing or completely avoiding activities that exacerbate symptoms is crucial initially. Transitioning to low-impact exercises such as cycling or swimming can help minimise foot strain while maintaining fitness.


Non-steroidal Anti-inflammatory Drugs (NSAIDs)

NSAIDs like ibuprofen can alleviate pain and reduce inflammation. However, they do not address the underlying issues associated with PCFD and some research suggests these drugs may compromise tendon healing, they should be used sparingly/with caution.


Orthotics and Bracing

Orthotic devices, either custom-made or over-the-counter, can support the foot's arch, alleviating pain and redistributing pressure on the foot. Ankle braces can also provide support, particularly in moderate cases.


Physical Therapy

A structured physical therapy program focusing on strengthening the PTT and stretching surrounding muscles may be beneficial for individuals with mild to moderate symptoms.


Steroid Injections

Although powerful anti-inflammatory injections can provide temporary relief, they carry risks, particularly in the PTT, where they could potentially lead to rupture.


Surgical Treatment Options

If conservative measures prove insufficient after several months, surgical intervention may be warranted. The specific surgical approach depends on several factors, including the severity of the deformity and associated issues such as arthritis. Various surgical procedures are available, ranging from simple tendon repair to more complex reconstructive surgeries, tailored to address the specific needs of each patient. An orthopaedic surgeon would be best placed to advise on these options.


Conclusion

Progressive collapsing foot deformity is a complex disorder that can severely impact quality of life. While it often begins as a subtle issue, failing to recognise and address the symptoms early can lead to significant complications. Through a comprehensive understanding of the condition and its treatment options, affected individuals can work with healthcare providers to implement effective management strategies, ensuring better outcomes and improved mobility.


Find Your Stride!

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